Read the latest news and updates from the Biology department.
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The Spring 2017 Biology Seminar Series continues with Gage Crump’s talk, "Using Zebrafish to Model Diseases of the Human Face and Skull."
The Spring 2017 Biology Seminar Series continues with Susan Piacenza’s talk, "Fathoming Sea Turtles: How to Improve Population Assessments of Unstable Populations."
The Spring 2017 Biology Seminar Series continues with Sergio Avila’s talk, "From fur to fuzz: protecting northern jaguars while studying monarch butterflies along the US-Mexico borderlands."Sponsored by the Biology Department and the Remsen Bird Fund
The Spring 2017 Biology Seminar Series continues with Sergio Avila’s talk, "From fur to fuzz: protecting northern jaguars while studying monarch butterflies along the US-Mexico borderla
The Spring 2017 Biology Seminar Series commences with David W. Scott's talk, "Driving CARs to BARs: The road to engineered human antigen-specific regulatory and cytotoxic T cells."
The emergence of infectious disease rarely ends in the complete extinction of host species. Frequently, the level of virulence in a severe disease system shifts such that hosts and pathogens can persist in a shared environment. However, the mechanistic underpinnings of these transitions are not well understood.
Sharon Stranford studies factors that influence the development of acquired immune deficiency. She and the undergraduate students who work in her research lab employ a mouse model of AIDS (MAIDS) in which some strains of mice develop immune deficiency following exposure to Murine Leukemia Virus (MuLV).
Trichomonas vaginalis is a protozoan parasite that causes the most common non-viral STI in the U.S. and worldwide. While complications due to Trichomonas infection can contribute to adverse reproductive outcomes and health disparities, not much is known about how the immune system responds to Trichomonas, or why the infection persists.
